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dc.contributor.advisorGoering, Richarden_US
dc.contributor.authorViola, Colleen Koziseken_US
dc.date.accessioned2017-10-05T21:21:02Z
dc.date.available2017-10-05T21:21:02Z
dc.date.issued1977en_US
dc.identifier.urihttp://hdl.handle.net/10504/114422
dc.description.abstractNumerous studies have demonstrated the existence of DNA repair systems which function to preserve the basic integrity of genetic material. These repair systems are of two basic classes: the first class contains a repair system which functions only in the presence of light (photoenzymatic repair); the second class includes those systems which function optimally in the dark (excision and recombinational repair).| Photoenzymatic repair is known to be specific for chromosomal lesions which have been induced by ultraviolet light. In contrast, the dark repair systems are error-correcting mechanisms which alleviate the damaging effects of chromosomal lesions produced by a wide variety of agents, including X-radiation, nitrosoguanidine, and mitomycin C. In addition, it has been reported that mutants deficient in either excision or recombinational repair produce significant numbers of non-viable cells. These observations support the role of DNA dark repair systems in the general maintenance of chromosomal DNA.en_US
dc.language.isoen_USen_US
dc.publisherCreighton Universityen_US
dc.rightsA non-exclusive distribution right is granted to Creighton University and to ProQuest following the publishing model selected above.en_US
dc.subjectMicrobiologyen_US
dc.titlePlasmid-Host Cell Relationships in Staphylococcus Aureus: The Role of Deoxyribonucleic Acid Dark Repair Systemsen_US
dc.typeThesis
dc.publisher.locationOmaha, Nebraskaen_US
dc.description.noteProQuest Traditional Publishing Optionen_US
dc.contributor.cuauthorViola, Colleen Koziseken_US
dc.degree.levelMS (Master of Science)en_US
dc.degree.disciplineMedical Microbiology (graduate program)en_US
dc.degree.nameM.S. in Medical Microbiologyen_US
dc.degree.grantorGraduate Schoolen_US


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